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Equine metabolic syndrome (EMS) is defined as a complex disorder characterised by a phenotype of insulin dysregulation, obesity and a predisposition toward laminitis, resulting from the combination of one or more inherited genetic alleles and environmental influences.1
Recently, a genome-wide association study (GWAS)2 identified two genetic markers for EMS located in the FAM174A gene region in Arabian horses: BIEC2-263524-C and FAM174A 3’ UTR -11(G) (Equus caballus chromosome 14:69,276,814 and 14:69,119,991 …
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